Nucleotide substitution: G?A. Mutation at the exon 7 splice acceptor site, resulting in a frameshift after amino acid residue 825.
G8068869A
G to A base change at the splice acceptor. Causes a frameshift after aa residue 825.
Homozygous somatic clones in the wing result in wing blisters. Mutant embryos show dorsal herniation.
ifunspecified, mysG1 has lethal | dominant phenotype
mys11/mysG1 is rescued by mysUbi-p63E.Venus
mys11/mysG1 is partially rescued by mysE810Q.E817Q.Ubi-p63E.Venus
mys11/mysG1 is not rescued by mysN828A.Ubi-p63E.Venus
mysE810Q.E817Q.Ubi-p63E.T:Avic\GFP-YFP.Venus fails to rescue the germband retraction and dorsal closure defects of mys11/mysG1 embryos, but partially rescues the myotendinous junction defects.