The Scer\FRT cassette has been excised, juxtaposing the Act5C promoter with sequences encoding a constitutively active form of Mmus\Pkaca.
Scer\FRT-y+t7.7 cassette has been excised, juxtaposing Act5C promoter with mutated Mmus\Pkaca coding sequences (mC*).
Mmus\PrkacamC.Act5C is a non-suppressor of lethal phenotype of Pka-C1H2
Mmus\PrkacamC.Act5C is a suppressor of nurse cell | germline clone phenotype of Pka-C1H2
Mmus\PrkacamC.Act5C is a suppressor of oocyte | germline clone phenotype of Pka-C1H2
Mmus\PrkacamC.Act5C is a suppressor of wing | somatic clone phenotype of Pka-C1H2
Mmus\PrkacamC.Act5C is a suppressor of scutum | somatic clone phenotype of Pka-C1H2
Mmus\PrkacamC.Act5C is a suppressor of haltere | somatic clone phenotype of Pka-C1H2
Mmus\PrkacamC.Act5C is a suppressor of antenna | somatic clone phenotype of Pka-C1H2
Expression of the constitutively active Mmus\PkacamC.Act5C transgene rescues the oocyte polarity and nurse cell fusion phenotypes in Pka-C1H2 germline clones. However, the transgene is unable to rescue the embryonic and larval lethality of Pka-C1H2 mutants.
Homozygous Pka-C1H2 female germ line clones that also carry Mmus\PkacamC.Act5C give rise to eggs which can be fertilised and develop in a small number of cases. Many Pka-C1H2/Df(2L)γ15 embryos derived from these germ line clones hatch, and have ventral cuticle patterns similar to wild-type.
Suppresses the majority of pattern defects caused by Pka-C1H2 clones in the wing, notum, halteres and antennae.
Exists in clones, not reported as being germ-line transmitted.