Clonal analysis indicates that the rate of proliferation in imaginal discs is reduced to approximately 63% of wild type in homozygous clones.
Twin spot analysis demonstrates a reduction of the proliferation rate. Anterior and posterior structures are defective in some embryos derived from homozygous germ line clones, others exhibit poor cuticle development.
Dsor1r1 has decreased occurrence of cell division | somatic clone | larval stage phenotype, suppressible by rlSu23/rl[+]
Dsor1r1 has lethal | recessive phenotype, non-suppressible by hepUbi-p63E.PG
Dsor1r1 has lethal | recessive phenotype, non-suppressible by Mmus\Map2k7Ubi-p63E.PH
Dsor1r1 has lethal | recessive phenotype, non-suppressible by Mmmm\Sek1Ubi-p63E.PH
Dsor1r1 has lethal | recessive phenotype, non-suppressible by Xlae\Mek2Ubi-p63E.PH
Dsor1r1 has imaginal disc | somatic clone | larval stage phenotype, suppressible by rlSu23/rl[+]
Dsor1r1/Scer\GAL4Bx-MS1096 is an enhancer of wing phenotype of Bant\lefUAS.cGa
Dsor1r1/Scer\GAL4Bx-MS1096 is an enhancer of wing vein phenotype of Bant\lefUAS.cGa
Dsor1r1 is a suppressor of embryonic/first instar larval cuticle | maternal effect phenotype of tor12D
Dsor1r1 is a suppressor of phenotype of Src42AKR.hs.2sev
The imaginal disc proliferation defect is suppressed by rlSu23/+.
The lethality of hemizygous males is not rescued by hepUbi-p63E.PG.
Moderate expression of Zzzz\lefScer\UAS.cGa under the control of Scer\GAL4Bx-MS1096 in Dsor1r1 results in an aggravated Zzzz\lefScer\UAS.cGa (Scer\GAL4Bx-MS1096) phenotype consisting of more pronounced scooped wings and distal vein truncations.
The lethality of hemizygous males is not rescued by Mmus\Prkmk7Ubi-p63E.PH, Mmmm\Sek1Ubi-p63E.PH or Xlae\Mek2Ubi-p63E.PH.
Lost the suppressing activity of Dsor1Su1.