UAS regulatory sequences drive expression of a short inverted repeat.
Expression of Ndc80GL00625 under the control of Scer\GAL4mat.αTub67C.T:Hsim\VP16 in oocytes does not significantly disrupt kinetochore assembly, prometaphase karyosome configurations, or co-orientation of homologous chromosomes, but does disrupt end-on kinetochore-microtubule attachment formation and centromere orientation towards the spindle pole.
Expression of Ndc80GL00625 under the control of Scer\GAL4nos.UTR.T:Hsim\VP16 in females results in sterility.
Ndc80GL00625, Scer\GAL4VP16.mat.αTub67C is an enhancer of karyosome phenotype of Scer\GAL4VP16.mat.αTub67C, cmetGL00404
Co-expression of Ndc80GL00625 enhances the karyosome splitting defect of oocytes expressing cmetGL00404 under the control of Scer\GAL4mat.αTub67C.T:Hsim\VP16 results in karyosome splitting.