Expression of ckGD1408 under the control of Scer\GAL4Act.PU in clones in the Johnston's organ does not lead to any defects in attachment of scolopidia to the hinge of the second and third antennal segment.
Scer\GAL4Act.PU/ckUAS.GFP is an enhancer | somatic clone of abnormal neuroanatomy | P-stage | somatic clone phenotype of Ubr3B
Scer\GAL4en-e16E/ckUAS.GFP is an enhancer of visible phenotype of Scer\GAL4en-e16E, zipRod.UAS.GFP
Scer\GAL4Act.PU/ckUAS.GFP is an enhancer | somatic clone of Johnston organ | P-stage | somatic clone phenotype of Ubr3B
Scer\GAL4Act.PU/ckUAS.GFP is an enhancer | somatic clone of scolopidium | P-stage | somatic clone phenotype of Ubr3B
Scer\GAL4en-e16E/ckUAS.GFP is an enhancer of wing hair | increased number phenotype of Scer\GAL4en-e16E, zipRod.UAS.GFP
The scolopidia detachment phenotype observed in Ubr3B mutant clones in the pupal Johnston's organ is significantly enhanced by expression of ckScer\UAS.T:Avic\GFP under the control of Scer\GAL4Act.PU in the mutant cells.
Co-expression of ckScer\UAS.T:Avic\GFP enhances the penetrance of the multiple wing hair phenotype caused by expression of zipRod.Scer\UAS.T:Avic\GFP under the control of Scer\GAL4en-e16E.
ckUAS.GFP/Scer\GAL4sqh.PW rescues ck13
ckUAS.GFP/Scer\GAL4nompA.cK partially rescues ck13
Scer\GAL4elav-C155/ckUAS.GFP partially rescues ck13
Expression of ckScer\UAS.T:Avic\GFP in the scolopale cells, driven by Scer\GAL4nompA.cK, or in neurons, driven by Scer\GAL4elav-C155, leads to a significant partial rescue of the ck13 auditory phenotype.