FB2024_03 , released June 25, 2024
Allele: Dmel\stan192
Open Close
General Information
Symbol
Dmel\stan192
Species
D. melanogaster
Name
FlyBase ID
FBal0143195
Feature type
allele
Associated gene
Dmel\stan
Associated Insertion(s)
Carried in Construct
Also Known As
fmi192
Key Links
Allele class

loss of function allele

Mutagen
Nature of the Allele
Allele class

loss of function allele

(Rawls and Wolff, 2003.4.28)
Mutagen
X ray
(Rawls and Wolff, 2003.4.28)
Progenitor genotype
Cytology
Description
Mutations Mapped to the Genome
Curation Data
Type
Location
Additional Notes
References
Variant Molecular Consequences
Associated Sequence Data
DDBJ / EMBL / GenBank
DNA sequence
Protein sequence
 
UniProtKB/Swiss-Prot
    UniProtKB/TrEMBL
      Expression Data
      Reporter Expression
      Additional Information
      Statement
      Reference
       
      Marker for
      Reflects expression of
      Reporter construct used in assay
      Human Disease Associations
      Disease Ontology (DO) Annotations
      Models Based on Experimental Evidence ( 0 )
      Disease
      Evidence
      References
      Modifiers Based on Experimental Evidence ( 0 )
      Disease
      Interaction
      References
      Comments on Models/Modifiers Based on Experimental Evidence ( 0 )
       
      Disease-implicated variant(s)
       
      Phenotypic Data
      Phenotypic Class
      Phenotype Manifest In
      Detailed Description
      Statement
      Reference

      stan192/stanE59 stage 16 embryos have longer dorsal trunks than normal.

      (Chung et al., 2009)

      When homozygous mutant somatic clones are made in the eye, tissue polarity defects are seen in the ommatidia.

      (Rawls and Wolff, 2003)
      External Data
      Interactions
      Show genetic interaction network for Enhancers & Suppressors
      Phenotypic Class
      Enhancer of
      Statement
      Reference

      stan[+]/stan192 is an enhancer of visible phenotype of DgRNAi.UAS, Scer\GAL4Tub.PU

      (Kucherenko et al., 2008)

      stan192 is an enhancer of abnormal planar polarity phenotype of Vangsev.PR

      (Rawls and Wolff, 2003)
      Suppressor of
      Statement
      Reference

      stan[+]/stan192 is a suppressor of abnormal planar polarity phenotype of Scer\GAL4hs.2sev, dgoUAS.cFa

      (Weber et al., 2012)

      stan[+]/stan192 is a suppressor of visible phenotype of DysRNAi.NH2.UAS, Scer\GAL4Act.PU

      (Kucherenko et al., 2008)

      stan[+]/stan192 is a suppressor of visible phenotype of DysRNAi.C.UAS, Scer\GAL4Tub.PU

      (Kucherenko et al., 2008)
      Phenotype Manifest In
      Enhancer of
      Statement
      Reference

      stan[+]/stan192 is an enhancer of posterior crossvein phenotype of DgRNAi.UAS, Scer\GAL4Tub.PU

      (Kucherenko et al., 2008)

      stan192 is an enhancer of ommatidium phenotype of Vangsev.PR

      (Rawls and Wolff, 2003)
      Suppressor of
      Statement
      Reference

      stan[+]/stan192 is a suppressor of ommatidium phenotype of Scer\GAL4hs.2sev, dgoUAS.cFa

      (Weber et al., 2012)

      stan[+]/stan192 is a suppressor of posterior crossvein phenotype of DysRNAi.NH2.UAS, Scer\GAL4Act.PU

      (Kucherenko et al., 2008)

      stan[+]/stan192 is a suppressor of posterior crossvein phenotype of DysRNAi.C.UAS, Scer\GAL4Tub.PU

      (Kucherenko et al., 2008)
      Other
      Statement
      Reference

      DysRNAi.NH2.UAS, stan[+]/stan192 has wing vein | ectopic phenotype

      (Kucherenko et al., 2008)

      DysRNAi.C.UAS, stan[+]/stan192 has wing vein | ectopic phenotype

      (Kucherenko et al., 2008)
      Additional Comments
      Genetic Interactions
      Statement
      Reference

      stan192/+ significantly suppresses the eye defects induced by the overexpression of dgoScer\UAS.cFa under the control of Scer\GAL4hs.2sev.

      (Weber et al., 2012)

      One copy of stan192 moderately suppresses the detached posterior crossvein phenotype seen when DysdsRNA.NH2.Scer\UAS is expressed under the control of Scer\GAL4Act.PU but produces extra wing vein material.

      One copy of stan192 weakly suppresses the detached posterior crossvein phenotype seen when DysdsRNA.C.Scer\UAS is expressed under the control of Scer\GAL4tub.PU but produces extra wing vein material.

      One copy of stan192 enhances the posterior crossvein phenotype seen when DgdsRNA.Scer\UAS is expressed under the control of Scer\GAL4tub.PU.

      (Kucherenko et al., 2008)
      Xenogenetic Interactions
      Statement
      Reference
      Complementation and Rescue Data
      Comments
      Images (0)
      Mutant
      Wild-type
      Stocks (1)
      Notes on Origin
      Discoverer
      External Crossreferences and Linkouts ( 0 )
      Synonyms and Secondary IDs (2)
      References (10)