Adult escapers of Moe^PL106 appear at low frequency. Wings of these escapers often appear crumpled (displaying cuticle vesicles trapped between the two wing surfaces) and reduced in size and their eyes display a rough-eye phenotype, because ommatidia are reduced in number and appear disorganized.

While expression of Moe^{TA.Scer\UAS.T:Avic\GFP-EGFP} using Scer\GAL4^αTub84B.PL fully rescues the wing phenotypes of Moe^PL106, it only partially rescues the viability, and fails to rescue the eye phenotypes.

Expression of Moe^{KN.Scer\UAS.P\T.T:Avic\GFP} using Scer\GAL4^αTub84B.PL slightly enhances the viability of Moe^PL106, however, the escapers display rough eyes and crumpled wings.

While expression of Moe^{KNTD.Scer\UAS.P\T.T:Avic\GFP} using Scer\GAL4^αTub84B.PL rescues the external wing phenotypes and enhances the viability of Moe^PL106, it fails to rescue the eye phenotypes.

Moe^PL106 mutant clones induce ectopic cell death in the wing disc epithelium.

Expression of Moe^{KNTD.Scer\UAS.P\T.T:Avic\GFP} under the control of Scer\GAL4^αTub84B.PL fails to rescue the ectopic cell death induced by somatic clones of Moe^PL106.

Wing discs from moe^PL106 homozygotes become progressively disorganised during growth. Early third instar discs show a more normal appearance but alterations in the basal lamina are already present at this stage. By late third instar, the disc is misfolded and has breaks in the basal lamina. Defects in the hinge region including mislocalisation of apical markers, misfolding folding and disorganisation of the basal lamina are more severe than those seen in Moe^c858 mutants. There is also an increase in cell death in the hinge region.

A proportion of the eggs laid by heterozygous Moe^PL106 heterozygous females display segmentation defects (12%, n=223). These abnormal embryos lack posterior structures. No sign of premature cytoplasmic streaming is seen in Moe^PL106 germline clone oocytes. However, defects are apparent in the actin cytoskeleton: microfilaments are more loosely bound to the posterior and lateral cortex of the oocyte than in wild-type, and are also sometimes found clumped together outside of the cortex. These defects are not seen in wild-type oocytes overlain by Moe^PL106 mutant follicle cells. In Moe^PL106 germline clone nurse cells, actin filaments are often absent from the cortex and concentrated at the cell perimeter. They also accumulate in abnormal structures at the point of contact between nurse cells.