fs(1)h^rnc hemizygotes have a cuticular phenotype, with disruptions to the head skeleton.

fs(1)h^rnc/fs(1)h¹ females are fertile at 18[o]C, but are unable to lay eggs at 22[o]C.

Progeny from fs(1)h^rnc/fs(1)h¹ mothers, raised at semipermissive temperatures (22[o]C), do not hatch. Cuticle analysis reveals that the second abdominal segment is deleted or fused in all of these animals, and most show fusion and mid-line defects in additional segments. The filzkorper is deleted or malformed in 95% of animals. Head structures are often indiscernible and occasionally absent altogether.

Homozygotes and hemizygotes show defects in derivatives of the antennal and ventral gnathal lobes, including Dfd-dependent structures. The hypostomal sclerite and H-piece arm are present but malformed, the base of the mouth hook is reduced, and the mouth hooks are multiply serrated. Head involution fails, causing misplacement of maxillary and antennal sense organs, similar to that caused by lab mutants.

fs(1)h^rnc is an enhancer of visible | adult stage phenotype of Hsap\MAPT^UAS.cWa, Scer\GAL4^GMR.PF

fs(1)h[+]/fs(1)h^rnc is an enhancer of visible | adult stage phenotype of Hsap\MAPT^UAS.cWa, Scer\GAL4^GMR.PF

fs(1)h[+]/fs(1)h^rnc is an enhancer of partially lethal - majority die phenotype of Dfd¹³/Dfd³

fs(1)h^rnc is an enhancer of eye phenotype of Hsap\MAPT^UAS.cWa, Scer\GAL4^GMR.PF

fs(1)h[+]/fs(1)h^rnc is an enhancer of eye phenotype of Hsap\MAPT^UAS.cWa, Scer\GAL4^GMR.PF