Polytene chromosomes normal.
11bp deletion, predicted to cause a frameshift after amino acid 78 and truncation of the protein after the addition of seven out-of-frame amino acids. Only the N-terminal 25% of the protein is predicted to remain.
11bp deletion, predicted to cause a frameshift after amino acid 78 and truncation of the protein after the addition of 7 novel amino acids.
rux8 mutant males show normal spermatocyte fusome morphology.
All cells within the morphogenetic furrow ectopically enter S phase.
Entry in the S phase is precocious, all cells reenter the cell cycle. Male germ cells progress through meiosis I and meiosis II normally but then undergo an ectopic round of nuclear division.
Ectopic S phase entry in the morphogenetic furrow. Neither a CycE deficiency or a strong point mutation dominantly suppresses the S phase phenotype.
No effect on the faf eye phenotype.
Eyes are rough, viability reduced to 10% with respect to wild type. Photoreceptor clusters form in rux8 with the same frequency and spacing as wild type. However, the organization of cells within the clusters is usually disrupted. The R8 cell number is often abnormal, and numbers of R1, R3, R4 and R6 are reduced.
rux8 has male sterile phenotype, suppressible by twe1/twe[+]
rux8 has male sterile phenotype, non-suppressible by stgAR2/stg[+]
rux8 has male sterile phenotype, non-suppressible by stgRXT13/stg[+]
rux8 is a suppressor of decreased occurrence of cell division | larval stage phenotype of CycEJP
rux8 is a suppressor of eye disc posterior to the morphogenetic furrow | larval stage phenotype of CycEJP