Heterozygous adults have wild-type wings.

The temperature sensitive heterozygous combination, Dl^vi1/Dl^6B leads to animals that exhibit a ISNb axon bypass phenotype. These axons reach their targets via an aberrant trajectory, in which ISNb axons remain associated with the ISN. About 23% of hemisegments are affected. The formation of neuromuscular synapses to ventral longitudinal muscles occur as efficiently in mutant animals than in controls. Other kinds of ISNb misrouting phenotypes are seen at a low frequency. However gross stalling of ISNb axons is not seen, nor are defects in muscle development.

Abnormal microtubule organisation is seen in Dl^vi1/Dl^6B oocytes.

Dl^6B/Dl^vi1 embryos show a great reduction in egg laying at the restrictive temperature (32^oC). Mislocalization of bcd to the posterior pole of the oocyte and excess of polar cells were evident.

Heterozygotes have extreme extra microchaetae phenotypes.

Homozygous adults exhibit a high penetrance of tarsal segments 3 and 4 fusion.

Homozygous viable. Slight δ-like thickenings at posterior tips of wing veins 2, 3, 4 and 5; roughening of eye. Dl^vi1 homozygotes also show shortening and frequent fusion of tarsal segments. A few homozygous embryos fail to hatch, showing patchy neuralization in cephalic and ventral territories. Dl^vi1/+ normal. Trans heterozygotes with dominant alleles show extreme wing, eye and tarsal abnormalities.

Delta^vi1, ed^m1/ed^ts has wing vein | ectopic phenotype

Delta^vi1, ed^m1/ed^ts has posterior cell phenotype

Delta^vi1/Dl[+], ed^m1/ed^ts has wing vein | ectopic phenotype

Delta^vi1/Dl[+], ed^m1/ed^ts has posterior cell phenotype