Nucleotide substitution: G?A.
Amino acid replacement: W63term.
G8561739A
G?A
W63term | rhea-PB; W63term | rhea-PC; W63term | rhea-PD; W63term | rhea-PE; W63term | rhea-PF; W63term | rhea-PG; W63term | rhea-PH; W63term | rhea-PI
W63term
Nonsense mutation (TGG to TGA) at Trp codon.
Homozygous clones in the wing result in wing blisters in more than 95% of wings.
Homozygous embryos show complete muscle detachment (quantified as greater than 40% shortening of the dorsal muscles).
More than 25% of homozygotes fail to undergo germband retraction (this phenotype is indistinguishable from that of a null mutation).
rheaB28 is a suppressor of follicle cell | somatic clone phenotype of Scer\GAL4Act5C.PU, VincCO.UASp.TagRFP
rheaB28 is a suppressor | partially of follicle cell | somatic clone phenotype of Scer\GAL4Act5C.PU, VincHead.UASp.TagRFP
rheaB28 is a suppressor | partially of follicle cell | somatic clone phenotype of Scer\GAL4Act5C.PU, VincD1.UASp.TagRFP
rheaB28 homozygosity nearly abrogates the ectopic VincCO.Scer\UAS.P\T.T:Equa\eqFP578-TagRFP-positive cytoplasmic protein aggregates formed in follicle cell clones and embryonic muscles, upon expression under the control of Scer\GAL4Act5C.PU and Scer\GAL4Mef2.PU, respectively.