SarmUAS.EGFP/Scer\GAL4VGlut-OK371 partially rescues Sarm896
The axon death defect (severed sensory neuron axons in the adult wing persist after an axotomy, instead of being cleared away) observed in flies carrying Ect4896 neuronal mutant clones can be partially rescued by Scer\GAL4VGlut-OK371-driven expression of Ect4Scer\UAS.T:Avic\GFP-EGFP in the mutant clones.