UASt regulatory sequences drive expression of an inverted repeat.
Stage 16-17 embryos expressing Ppt1GD3286 under the control of Scer\GAL4elav.PU exhibit a range of axon guidance phenotypes including wavy bundles with some faulty axon crossing over the midline. Similar phenotypes are seen when Ppt1GD3286 is expressed under the control of either eve2xCQ.RC.T:Scer\GAL4 or Scer\GAL4eve.RN2.
Expressing Ppt1GD3286 under the control of Scer\GAL4GMR.PU induces light-dependent ommatidial degeneration (ommatidium disruption and rhabdomere loss), associated with retinal apoptosis.
Adults expressing Ppt1GD3286 under the control of Scer\GAL4elav.PLu (in the presence of Dcr-2Scer\UAS.cDa to increase the efficiency of RNAi) do not show a significant defect in avoidance of noxious temperature (46[o]C) compared to control flies.
Ppt1GD3286, Scer\GAL4GMR.PU is an enhancer of ommatidium | adult stage | light conditional phenotype of PisdNIG.5991R, Scer\GAL4GMR.PU
Ppt1GD3286, Scer\GAL4GMR.PU is an enhancer of rhabdomere | adult stage | light conditional | decreased number phenotype of PisdNIG.5991R, Scer\GAL4GMR.PU