Imprecise excision of the P{lacW} from ash2S112411 has led to a 2bp deletion and 5bp insertion in the fourth intron of ash2.
Imprecise excision of the P{lacW} from ash2S112411 has led to a 2bp deletion (GT) and 5bp insertion (TTAGG) in the fourth intron of ash2.
Mutant wing discs show a high concentration of dying cells in the prospective intervein regions of the wing pouch.
Late third instar wing discs in ash2I1 homozygotes are much smaller than wild-type. minute -/-; ash2I1 homozygous somatic clones in the wing in a minute -/+ background show intervein reduction and extra vein tissue, preferentially close to the normal veins, which appear thickened.
The loss of anterior cross-vein and L4 gap seem in Egfrf11/Egfrt1 wings are partially rescue by ash2I1/+.
Homozygous mutants die as pupae, and have reduced and abnormal imaginal discs.
When mutant wing clones are made in a Minute background, the resulting wings are distorted, show blistering, with strongly reduced intervein regions. When a clone runs over a wing vein (all except L4), thickening is seen.
ash2[+]/ash2I1 is a suppressor | partially of anterior crossvein phenotype of Egfrt1/Egfrf11
ash2[+]/ash2I1 is a suppressor | partially of wing vein L4 phenotype of Egfrt1/Egfrf11
Formation of ectopic vein tissue in rlSem/+ animals is enhanced by ash2I1/+.
net1 homozygotes develop extra vein tissue, mainly in the form of ectopic transverse connections between L2 and L3 and between L4 and L5. This phenotype is enhanced by ash2I1/+ which also have a reduction in the size of the intervein regions between L2 and L3 (submarginal cell) and between L4 and L5 (discal cell and second posterior cell). The resulting wings have a lanceolate shape.
