Insertion of a P{lacW} element.
Homozygous embryos exhibit pathfinding and connectivity defects, mainly from the dorsal to lateral cluster. Five lateral chordotonal (Lch) neurons are stalled along the dorsoventral path of their migration, close to their final destination.
I. Kiss.
Deficiency complementation analysis is consistent with the lethality of the line being associated with the insertion.
P{lacW} insertion can be excised and revert the lethal phenotype to wild type.