Embryos from swa¹/swa³ females have a reduced embryonic head.

Embryos derived from swa³/Df(1)JF5 females mated to wild-type males often show cuticle defects, ranging from embryos without cuticle and lacking any sign of development (57%), through embryos with a reduction in the number of abdominal segments to embryos that have 8 abdominal segments, with normal or nearly normal abdominal morphology. The most commonly represented cuticular class in embryos that develop is embryos that have 4-6 abdominal segments, with the arrangement of segments usually abberant, with segments often fused (43%).

Does not prevent the posterior localization of G-iα65A protein.

Pregastrula and gastrula embryos exhibit nuclear defects, an excess of irregularly shaped internal chromatin masses, blastoderm nuclei are non-uniform in size and chromatin condensation and asynchronous in the cell cycle. Excess of irregularly shaped internal chromatin masses, and chromatin condensation is asynchronous. During preblastoderm and early blastoderm cleavages lagging chromosomes during anaphase are common.

Strong allele.

Embryonic defects detectable at gastrulation. Eggs are laid that collapse on deposition.

maternal-effect lethal strong allele female-sterile.

swa³/swa¹ is a suppressor of visible | homeotic phenotype of mira^{Δ103.αTub67C.mGFP6}

swa³/swa[+] is a suppressor of female sterile | semidominant phenotype of αTub67C³

swa³/swa¹ is a suppressor of embryonic abdomen | ectopic phenotype of mira^{Δ103.αTub67C.mGFP6}

swa³/swa¹ is a suppressor of embryonic thorax phenotype of mira^{Δ103.αTub67C.mGFP6}

swa³/swa[+] is a suppressor of embryonic/first instar larval cuticle | maternal effect phenotype of αTub67C³