wgGla-1 mutant flies do not display any wing margin defects (nicks or notches).
Almost no photoreceptor cell structures are seen in the eye in heterozygotes. Most of the eye appears to consist of pigment cells and there is an excess of pigment granules. The overall distribution of pigment granules forms a faint regular pattern reminiscent of the arrangement of ommatidia in wild-type eyes. wgGla-1/wgrO216 animals develop into viable adults.
Abdomen of heterozygous female fails to expand with eggs.
Malpighian tubule colour: somewhat lighter than wild type; difficult to classify.
Abdomen of heterozygous female fails to distend with eggs; Eye reduced to one-fourth normal area and narrowed to a point ventrally; ommatidia coalesce into gleaming, smooth sheet. Eye colour: generally diluted but with some black patches.
jigr12205, wg[+]/wgGla-1 has visible | adult stage phenotype
The slightly reduced eye size and moderate wing margin nicking observed in the temperature-sensitive Nnd-1 homozygous adults at 18[o]C is enhanced by wgGla-1 heterozygosity: the ventral half of the adult eye is virtually completely lost and the wing notching phenotype is exacerbated too.
About 2/3 of wgGla-1/+;jigr12205/jigr12205 adult flies display a wing nicking phenotype (not observed in either of the single mutants alone), in rare cases very strong with extreme notches.
Induced on: In(2L)t. Associated with: In(2LR)Gla.
Associated with: In(2LR)Gla. The wgGla-1 mutation has been believed to be caused by one of the breakpoints of In(2LR)Gla, however evidence suggests that it is not associated with either breakpoint but is caused by the insertion of a roo element 1.5kb upstream of the wg coding region.
