34C1;35C1
34C1;35C1-35C2
34B12-34C1;35B10-35C1
34D3;35B10
34C1-34C2;35B10-35C1
bk1 << kuz << vas << bk2 << stc
This deletion removes a single ribosomal protein-coding gene (though other genes are also removed).
Inferred to overlap with: Df(2L)BSC345.
Flies heterozygous for the deletion do not show a Minute bristle phenotype.
Lethal in combination with: Df(2L)BSC147. Inferred to overlap with: Df(2L)BSC147.
Df(2L)b87e25 in combination with a pair of introgressions from D.simulans spanning 30F1-31E7 to 35D7-36A14 Dsim\Int(2L)S and 21A1 to 22D1--23A2 Dsim\Int(2L)D produces semi-sterile male flies. These flies are female sterile.
Suppresses the ommatidial polarity phenotype of dshhs.sev.B. Suppresses the ommatidial polarity phenotype of msnEP549 when driven by Scer\GAL4hs.2sev.
No second site non-complementing phenotype with zipEbr and zipmhc-c6.1.
Shows no maternal enhancement of dpphr4.
Deficient embryos show an uninterpretable mutant midgut phenotype.
Homozygous embryos are very abnormal compared to wild-type.
The stock contains a second-site deletion uncovering l(2)gl.
The Df(2L)b87e25 chromosome acts as a dominant weak suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{wvar}KR3-2", a stable "brown-red" variant of the P{3'WP-2,wvar}2Lt insertion), but this is a false positive result (the suppressor is not within the bounds of the deficient region) because the suppressor maps to the tip of 2L rather than to the site of the deficiency. In addition, the deficiency chromosome fails to complement lethal mutations of l(2)gl.
"irradiation" was stated as tentative. "spontaneous" was stated as tentative.
Ref: Lindsley and Zimm, 1992
Limits of break 1 from polytene analysis (FBrf0053558) Left limit of break 2 from inclusion of vas (FBrf0077941) Right limit of break 2 from polytene analysis (FBrf0080145)