The following information accompanied stocks donated to the Bloomington Stock Center by Nick Brown, University of Cambridge. Because Fit1 and Fit2 are at least partially redundant, individual mutations in Fit1 or Fit2 do not produce blisters in homozygous wing clones produced by mitotic recombination, but blisters are produced when 3L chromosome arms carrying Fit1 and Fit2 mutations are made homozygous in wing clones. To screen for other mutations involved in wing blister formation, Fit283 P{FRT(whs)}2A chromosomes were mutagenized with EMS and 3L chromosome arms were made homozygous in the presence of a vestigial-GAL4 insertion and a UAS-FLP insertion to look for wing blisters. The following mutations were isolated: hobC55 W2218X hobC236 W1985R ClC-cA27 L756X ClC-cB87 W338X Rcc1A47 T266I Rcc1C226 T266I Usp5A10 G781N Usp5C235 C207Y sflA7 Q426X sflA84 V477E sflC1 H993R gigasA25 C480Y and Y1662X gigasC28 V1602Q CG1136C227 K80X CG1136C245 Q25X rheaA16 rheaA109 rheaC63 rheaC110 rheaC130 rheaC208 Note that Rcc1A47 and Rcc1C266, although identical, are independent mutational events. In addition, two mutations in an unmapped complementation group were isolated. The locus was named 'crisp packet' (cpkt) as wings with clones induced by vg>FLP look like a crumpled-up potato chip/crisp packet. The alleles are: cpktA42 cpktC249