Contains a stop mutation near the translational start site.
DIP-β1-95 homozygous mutant adults have significantly increased numbers of synapses (Brp) in lamina cartridges, distally, proximally and overall, compared to controls; heterozygotes also have a significant increase distally and overall, but not proximally, compared to controls; L4 neurons of homozygotes have significantly increased distal, but not proximal or total, synapses on both primary dendrites and axon shafts, and significantly longer primary dendrites, compared to controls.
DIP-β1-95 has abnormal neuroanatomy | dominant | adult stage phenotype, suppressible | partially by DIP-γ1-67
DIP-β1-95, DIP-γ1-67 has abnormal neurophysiology | adult stage phenotype
DIP-β1-95, DIP-γ1-67 has abnormal optomotor response | adult stage phenotype
DIP-β1-95, DIP-γ1-67 has abnormal phototaxis | adult stage phenotype
DIP-β1-95, DIP-γ1-67 has increased rate of movement | adult stage phenotype
DIP-β1-95 has synapse | increased number | adult stage phenotype, suppressible | partially by DIP-γ1-67
DIP-β1-95 has optic cartridge phenotype, suppressible | partially by DIP-γ1-67
DIP-β1-95 has lamina monopolar neuron L4 phenotype, non-suppressible by DIP-γ1-67
DIP-β1-95 has dendrite | adult stage phenotype, non-suppressible by DIP-γ1-67
Young (1-2 day), but not older (13-15 day), DIP-β1-95 and DIP-γ1-67 double mutant adults have significantly increased tracking of optomotor stimuli and significantly increased attraction to light, compared to controls; older, but not younger, adults have significantly increased average speed, compared to controls; there are significant differences in their electroretinogram on and off transients and sustained components, compared to controls.