UAS regulatory sequences drive expression of a kinase-defective Abl::Hsap\ABL1::Hsap\BCR.
Expression of Abl::Hsap\ABL1::Hsap\BCRKR.Scer\UAS causes inappropriate crossovers of axon bundles in 4% or 42% of embryos when expressed using Scer\GAL4ftz.ng or Scer\GAL4elav.PLu, respectively.
Abl::Hsap\ABL1::Hsap\BCRKR.UAS/Scer\GAL4ftz.ng is an enhancer of abnormal neuroanatomy | embryonic stage 16 | dominant phenotype of robo11
Abl::Hsap\ABL1::Hsap\BCRKR.UAS/Scer\GAL4elav.PLu is an enhancer of abnormal neuroanatomy | embryonic stage 16 | dominant phenotype of sli1
Abl::Hsap\ABL1::Hsap\BCRKR.UAS/Scer\GAL4ftz.ng is an enhancer of larval longitudinal connective | embryonic stage 16 phenotype of robo11
Abl::Hsap\ABL1::Hsap\BCRKR.UAS/Scer\GAL4elav.PLu is an enhancer of larval longitudinal connective | embryonic stage 16 phenotype of sli1
Expression of Abl::Hsap\ABL1::Hsap\BCRKR.Scer\UAS using Scer\GAL4ftz.ng enhances the midline-crossing phenotype of axon bundles in heterozygous robo1 embryos.
Expression of Abl::Hsap\ABL1::Hsap\BCRKR.Scer\UAS using Scer\GAL4elav.PLu enhances the midline-crossing phenotype of axon bundles in heterozygous sli1 embryos.