UAS regulatory sequences drive expression of a short inverted repeat.
Expression of Dhc64CHMS01587 under the control of Scer\GAL4dpr1-PGaw strongly reduces the number of mitochondria that undergo axonal transport in both anterograde and retrograde directions in the wing marginal nerve, as compared to controls.
Dhc64CHMS01587, Scer\GAL4109(2)80 has mitochondrion | larval stage phenotype, non-enhanceable by Sod1G85R
Dhc64CHMS01587, Scer\GAL4109(2)80 has larval multidendritic neuron phenotype, non-enhanceable by Sod1G85R
Dhc64CHMS01587, Scer\GAL4109(2)80 has axon | larval stage phenotype, non-enhanceable by Sod1G85R
Dhc64CHMS01587, Scer\GAL4109(2)80 has mitochondrion | larval stage phenotype, non-suppressible by Sod1G85R
Dhc64CHMS01587, Scer\GAL4109(2)80 has larval multidendritic neuron phenotype, non-suppressible by Sod1G85R
Dhc64CHMS01587, Scer\GAL4109(2)80 has axon | larval stage phenotype, non-suppressible by Sod1G85R
Dhc64CHMS01587, Scer\GAL4VP16.mat.αTub67C is a suppressor | maternal effect of centrosome | maternal effect | embryonic stage phenotype of Scer\GAL4VP16.mat.αTub67C, aPKCHMS01320
Co-expression of Dhc64CHMS01587 rescues the abnormal centrosome positioning of embryos from mothers expressing aPKCHMS01320 under the control of Scer\GAL4mat.αTub67C.T:Hsim\VP16.