wing (with Df(3R)swp2MICAL)
wing (with Glut4EF10477)
wing (with Glut4EFEY03156)
wing (with Glut4EFEY04651)
wing (with Glut4EFNP7418)
wing (with Glut4EFsep)
Glut4EFNP7418/Glut4EFAntp-B adults show a 100% defect in wing positioning.
8% of heterozygotes have a held-out wings phenotype.
Glut4EFAntp-B, tna[+]/tna1 has visible | dominant phenotype
Glut4EFAntp-B, tna[+]/tna1 has wing phenotype
75% of Glut4EFAntp-B/tna1 flies have a held-out wings phenotype.
Glut4EFNP7418/Glut4EFAntp-B is rescued by Glut4EFd.UAS/Scer\GAL4NP7418
Expression of Glut4EFd.Scer\UAS under the control of Scer\GAL4Glut4EF-NP7418 rescues the wing positioning defects of Glut4EFNP7418/Glut4EFAntp-B flies.
The wing positioning defects in In(3R)AntpB flies were previously attributed to an effect on Antp (FBrf0107412 and FBrf0155711), however, FBrf0204343 demonstrates that these phenotypes are instead due to an effect on Glut4EF, which is also disrupted by the In(3R)AntpB chromosome.
FlyBase curator comment: FBrf0155711 attributes the wing positioning defects in In(3R)AntpB flies to an effect on Antp. However, FBrf0204343 demonstrates that the Glut4EF gene is also disrupted by the In(3R)AntpB chromosome, and that the wing positioning defects seen are instead due to an effect on Glut4EF.
FlyBase curator comment: FBrf0107412 attributes the wing positioning defects in In(3R)AntpB flies to an effect on Antp. However, FBrf0204343 demonstrates that the Glut4EF gene is also disrupted by the In(3R)AntpB chromosome, and that the wing positioning defects seen are instead due to an effect on Glut4EF.