UASt regulatory sequences drive expression of a 499bp inverted repeat.
Compared with wild-type, an increased number of unfused myoblasts are detected in embryos expressing Mp20NIG.4696R under the control of Scer\GAL4how-24B.
Compared with wild-type, expression of Mp20NIG.4696R under the control of Scer\GAL4how-24B results in the number of nuclei in muscles DT1 and VA2 decreased by about 2, while a lower decrease is detected in SBM.
Expression of Mp20NIG.4696R under the control of Scer\GAL4how-24B leads to a reduced rate of embryonic myoblast fusion (relative to wild-type) measured in VA2.
Embryos expressing Mp20NIG.4696R under the control of Scer\GAL4how-24B show a myoblast fusion defect characterised by rounded cells close to the segment border muscle.
Mp20NIG.4696R, Scer\GAL4how-24B is an enhancer of dorsal transverse muscle cell phenotype of PaxNIG.18061R, Scer\GAL4how-24B
Mp20NIG.4696R, Scer\GAL4how-24B is an enhancer of muscle cell of ventral acute muscle 2 phenotype of PaxNIG.18061R, Scer\GAL4how-24B
Mp20NIG.4696R, Scer\GAL4how-24B is an enhancer of nucleus phenotype of PaxNIG.18061R, Scer\GAL4how-24B
Co-expression of Mp20NIG.4696R and PaxNIG.18061R under the control of Scer\GAL4how-24B induces a strong decrease in the number of DT1 and VA2 nuclei, two muscles affected by single RNAi knockdowns. In contrast, the phenotype of single knockdowns in SBM is not enhanced by the double knockdowns.