uspunspecified mutants exhibit a severe neuronal pruning phenotype.
Homozygous mutant clones in the wing disc lead to premature differentiation of triple row bristle precursors, and disorganised ommatidia.
Border cells that are homozygous for uspunspecified in mosaic egg chambers show inhibition of border cell migration, but no obvious defects in other follicle cells.
uspunspecified has abnormal neuroanatomy phenotype, non-enhanceable by Hr39C105
uspunspecified has abnormal neuroanatomy phenotype, non-enhanceable by ftz-f1β.UAS.cBa/Scer\GAL4Tab2-201Y
uspunspecified has abnormal neuroanatomy phenotype, non-suppressible by Hr39C105
uspunspecified has abnormal neuroanatomy phenotype, non-suppressible by ftz-f1β.UAS.cBa/Scer\GAL4Tab2-201Y
uspunspecified has gamma Kenyon cell phenotype, non-enhanceable by Hr39C105
Hr39C105, uspunspecified has gamma Kenyon cell phenotype, non-enhanceable by ftz-f1β.UAS.cBa/Scer\GAL4Tab2-201Y
uspunspecified has gamma Kenyon cell phenotype, non-suppressible by Hr39C105
Hr39C105, uspunspecified has gamma Kenyon cell phenotype, non-suppressible by ftz-f1β.UAS.cBa/Scer\GAL4Tab2-201Y
uspunspecified Hr39C105 double mutants exhibit a severe neuronal pruning phenotype.
Expression of ftz-f1β.Scer\UAS.cBa in uspunspecified Hr39C105 double mutant neuroblast clones does not suppress the severe neuronal pruning phenotype observed in these mutants.