Mutation within the fourth intron (TG to AG) resulting in a new splice acceptor site (this mutation is in addition to the wgCE7 lesion on which wgPE1 was induced).
Mutation within the fourth intron (TG to AG) resulting in a new splice acceptor site (this mutation is in addition to the wgCE7 lesion on which wgPE1 was induced). The resulting protein includes 5 novel amino acids from intron sequences.