dpp^H61/dpp^H48 embryos show loss of dorsal tissue and expansion of the lateral denticle bands into dorsal regions. Head involution defects result in extruded globular structures at the anterior of the embryo.

Entire larval cuticle is ventralised. In dpp^H48; CrebA^wR7 larvae the entire cuticle is lateralised.

Heterozygotes produce a variable number (0 to 150) of amnioserosa cells. The average number of amnioserosa cells produced is not altered if the embryos are also carrying 2 or 3 copies of tsg^twi.PM.

Homozygous clones induced in first instar larvae produce abnormalities in the dorsal side of the leg. Pattern elements from the dorsal side are missing, and may or may not be replaced with a duplicate version of the remaining ventral part of the leg. Duplications are arranged as mirror images. Cells from non-mutant territory surrounding the clone may also show defects, indicating that dpp acts non-autonomously.

Double mutants of dpp^H48 with sog^YL26 or sog¹ have phenotypes identical to that of the dpp mutant alone.

Despite effects on the global morphology of the embryo the proventriculus still forms. Compared to wild type more cells move into the proventriculus and an extra budding zone in the foregut epithelium near the proventriculus can be seen in some embryos.

Fail to complete germ band extension, die with strong ventralized phenotype and no dorsal derived epidermis.

Homozygotes have a ventralized embryonic phenotype: normally ventral epidermal pattern elements are present ventrally and dorsally.