79E1-79E2;79E1-79E8
79E;79E
bk1 << Ten-m << bk2 << l(3)00506
Homozygous lethal.
The Df(3L)Ten-m-AL1 chromosome does not act as a dominant suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{wvar}KR3-2", a stable "brown-red" variant of the P{3'WP-2,wvar}2Lt insertion).
The segmentation defects previously reported to be due to the Df(3L)Ten-m-AL1 mutation are not due to this chromosome, but are instead are caused by a mutation in the opa gene (represented in by the oparSS allele) present on the TM3-rSS balancer in the original stock.
FlyBase curator comment: FBrf0214744 characterises new loss of function mutations in Ten-m and finds no evidence of a pair-rule segmentation phenotype, in contrast to previous reports. The authors show that the pair-rule phenotype previously reported for a number of Ten-m mutations (including Ten-m05309) was in fact due to a mutation present on the balancer in the original stock. The segmentation phenotype described in FBrf0098973 for Df(3L)Ten-m-AL1 has thus been removed from the aberration report in FlyBase, since it is not due to the mutation in Ten-m.
Left limit of break 1 from polytene analysis (FBrf0098973) Right limit of break 1 from inclusion of Ten-m (FBrf0098973) Left limit of break 2 from inclusion of Ten-m (FBrf0098973) Right limit of break 2 from polytene analysis (FBrf0098973)