48B-48C;58D-58E
48B-48C;58D-58E
bk1 << Adv << bk2
Inferred to overlap with: In(2R)vgW.
While complementation data indicate that the recessive lethality of In(2R)Adv is due to disruption of en, en expression appears normal in heterozygotes, suggesting that the dominant "additional vein" phenotype of this inversion is not due to its effects on en. Note - in situ hybridisation of polytene chromosomes with en cDNA also supports disruption of en by one of the breakpoints.
All limits from polytene analysis (FBrf0053664)