FB2024_03 , released June 25, 2024
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Bellen, H. (2017.1.20). PBac{fh.+3.3} insertions. 
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FBrf0234522
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The following information accompanied stocks donated to the Bloomington Drosophila Stock Center by Hugo Bellen at the Baylor College of Medicine.
PBac{fh.+3.3}VK00033 is a third chromosome insertion into the PBac{y+-attP-3B}VK00033 target site at 65B2 (R6,  3L:6442676 ), homozygous viable and fertile.
PBac{UAS-fh.ΔMTS.V5}VK00033 is a third chromosome insertion into the PBac{y+-attP-3B}VK00033 target site at 65B2 (R6,  3L:6442676 ), homozygous viable and fertile.
PBac{UAS-fh.V5}VK00033 is a third chromosome insertion into the PBac{y+-attP-3B}VK00033 target site at 65B2 (R6,  3L:6442676 ), homozygous viable and fertile.
PBac{UAS-hFXN.C}VK00033 is a third chromosome insertion into the PBac{y+-attP-3B}VK00033 target site at 65B2 (R6,  3L:6442676 ), homozygous viable and fertile.
These constructs are reported in Chen et al. (2016). Loss of Frataxin induces iron toxicity, sphingolipid synthesis, and Pdk1/Mef2 activation, leading to neurodegeneration.  eLife 5(): e16043.
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Loss of Frataxin induces iron toxicity, sphingolipid synthesis, and Pdk1/Mef2 activation, leading to neurodegeneration.
Chen et al., 2016, eLife 5: e16043 [FBrf0232938]

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