Embryos expressing FoxL1^Scer\UAS.cHa under the control of Scer\GAL4^twi.PG do not show any apparent hindgut morphological defects but display high frequency of irregularities in the structure of embryonic salivary gland - the glands are longer than in controls and the individual cells appear stretched (this is likely a secondary indirect consequence of defects in the gut development as assessed by the expression pattern of specific transcription factors) and placement aberrations in migratory cells/tissues - germ cells and Malpighian tubules. Salivary gland defects are also observed with the Scer\GAL4^{kirre-rP298-G4} or the Scer\GAL4^sns.PK drivers. Expression under none of the above drivers however leads to any strong muscle phenotype, although mild irregularities in muscle fusion are observed with Scer\GAL4^twi.PG. Expression of FoxL1^Scer\UAS.cHa controlled by the Scer\GAL4^fkh.PH driver leads to significant widening of the embryonic salivary gland.