Expression of trkN.Scer\UAS.P\T.T:Hsap\MYC,T:Disc\RFP-mCherry driven by Scer\GAL4nos.UTR.T:Hsim\VP16 results mostly in wild type embryos, though a small percentage (3%) show weak terminal patterning defects (loss of posterior filzkorper).
Scer\GAL4VP16.nanos.UTR, trkN.UASp.Tag:MYC,mCherry has embryonic/larval segmentation phenotype phenotype, enhanceable by Fur1GL01340, Scer\GAL4VP16.nanos.UTR
Scer\GAL4VP16.nanos.UTR, trkN.UASp.Tag:MYC,mCherry has embryonic/larval segmentation phenotype phenotype, enhanceable by Fur2GLC01802, Scer\GAL4VP16.nanos.UTR
Scer\GAL4VP16.nanos.UTR, trkN.UASp.Tag:MYC,mCherry has filzkorper phenotype, enhanceable by Fur1GL01340, Scer\GAL4VP16.nanos.UTR
Scer\GAL4VP16.nanos.UTR, trkN.UASp.Tag:MYC,mCherry has filzkorper phenotype, enhanceable by Fur2GLC01802, Scer\GAL4VP16.nanos.UTR
Co-expression of Fur1GL01340 or Fur2GLC01802 significantly enhances terminal patterning defects in Scer\GAL4nos.UTR.T:Hsim\VP16>trkN.Scer\UAS.P\T.T:Hsap\MYC,T:Disc\RFP-mCherry larvae.