In seq5 mutant homozygous clones, a large proportion of R7 cells terminate their axon projections in the outer M1-M3 layers, rather than the M6 layer. seq5 mutant R8 axons frequently stop above their prospective M3 target layer. When homozygous seq5 mutant clones are induced specifically in the R7 cells, almost all of the R7 axons stop prematurely in M3, even when surrounded by heterozygous R8 and R7 axons.