UAS regulatory sequences drive expression of a short inverted repeat.
Embryos expressing RhoGEF2HMS01118 under the control of Scer\GAL4VP16.nos.UTR show decreased contractile ring constriction in mid and late cellularization stages.
Wounded embryos expressing RhoGEF2HMS01118 under the control of Scer\GAL4mat.αTub67C.T:Hsim\VP16 show wound overexpansion, delayed actin accumulation around the wound edge and delayed repair, as compared to wounded control embryos.
RhoGEF2HMS01118, Scer\GAL4VP16.nanos.UTR has actomyosin contractile ring | maternal effect | embryonic stage 5 phenotype, suppressible by GrafCR57/GrafCR57
RhoGEF2HMS01118, Scer\GAL4VP16.nanos.UTR has actomyosin contractile ring | maternal effect | embryonic stage 5 phenotype, suppressible by GrafHMC03427, Scer\GAL4VP16.nanos.UTR
Scer\GAL4VP16.nanos.UTR/RhoGEF2HMS01118 is a suppressor of actomyosin contractile ring | maternal effect | embryonic stage 5 phenotype of GrafCR57
RhoGEF2HMS01118, Scer\GAL4VP16.nanos.UTR is a suppressor of actomyosin contractile ring | maternal effect | embryonic stage 5 phenotype of GrafHMC03427, Scer\GAL4VP16.nanos.UTR
RhoGEF2HMS01118, Scer\GAL4Act5C.PI, l(2)glGD4047 has wing disc | somatic clone | third instar larval stage phenotype
The clonal co-expression of either l(2)glGD4047 and RhoGEF2HMS01118 or scribKK101128 and RhoGEF2HMS01118 under the control of Scer\GAL4Act5C.PI (together with Dicer-2, for efficient RNAi) in third instar larval wing discs leads to a significant proportion of apical delamination, which does not occur upon l(2)glGD4047 or scribKK101128 single expression clones.
The clonal co-expression of scribKK101128, RhoGEF2HMS01118 and Stat92EΔNΔC.UASp.Tag:HA under the control of Scer\GAL4Act5C.PI (together with Dicer-2, for efficient RNAi) in third instar larval wing discs leads to overgrowths in the wing pouch domain, which does not occur upon scribKK101128 single expression or scribKK101128, RhoGEF2HMS01118 double expression.