When expression of l(1)scScer\UAS.cCa is driven by Scer\GAL4W381 the mutant muscle 25 phenotype of Df(1)sc19 is substantially rescued. The CNS defect of Df(1)sc19 is not rescued. In an otherwise wild type background there is a variable phenotype in which muscles are duplicated. The expressivity of this muscle duplication is increased by driving expression of l(1)scScer\UAS.cCa by Scer\GAL4how-24B Scer\GAL4twi.PG.
When E(spl)Scer\UAS.cNa and l(1)scScer\UAS.cCa are simultaneously driven by Scer\GAL4da.G32, neural hypoplasia effects are much weaker than those caused by E(spl)Scer\UAS.cNa alone, especially in the PNS. When E(spl)Scer\UAS.cNa and l(1)scScer\UAS.cCa are simultaneously driven by Scer\GAL4sca-537.4, ectopic sense organs caused by l(1)scScer\UAS.cCa are much reduced.
Scer\GAL4sca-4512-mediated expression fails to rescue the MP2 phenotype of homozygous In(1)y3PLsc8R embryos.