Insertion of approximately 8kb into the 3' UTR.
6.2 kb (3S18) insertion
abnormal size (with BxA523)
wing margin (with BxA523)
Mutant animals exhibit an decreased sensitivity to cocaine. After exposure to 100ug of cocaine, control animals show a slow circling behaviour and some are akinetic, mutant animals are much less affected, showing increased locomotion (mostly in straight lines) decreased slow circling, and almost no akinesia. Mutant animals, unlike controls which show very little movement, remain in motion at 125ug exposure showing circling behaviours.
Homozygotes exhibit tissue loss from the entire wing margin and blistering of the wing blade. The phenotype is unchanged in double homozygote combinations with ct53d.
The only dominant Bx allele not suppressed by a Bx deficiency or hdp.
Clonal analysis of wing disk development indicates massive cell loss during third larval instar. Clones of Bx+ cells in BxJ/+ wings that reach the margin but are confined to the dorsal or ventral surface often cause reconstitution of both surfaces and appearance of marginal elements derived from both surfaces.
Wings reduced to slender strip; only posterior cell present at tip. Heterozygous females have half and homozygous females one third the normal number of cells in membrane of wing. Femur shortened or legs otherwise abnormal, especially third pair. Halteres abnormal. Interacts with bi to give more nearly normal wings. RK1.
BxJ has wing phenotype, enhanceable by SsdpBG01663
BxJ has wing phenotype, enhanceable by SsdpKG03600
The addition of SsdpKG03600, SsdpBG01663, Ssdpneo48, Ssdp31 or Ssdp11 enhances the wing scalloping phenotype seen in BxJ animals.
BxJ is rescued by BxGD1385/Scer\GAL4He.PZ
Jollos, 1930.
"heat-treatment" was stated as tentative.
Alleles fall into an allelic series based on cocaine resistance phenotype. BxJ > Bx1 = Bx2 = Bx3.