A set of ~50 targeted deficiencies created by exploiting hybrid element insertion (HEI) and resolution; designed to fill gaps in deletion coverage.
66B12-66C14;66D2-66D4
66B12-66C1;66D2-66D4
Lethal in combination with T(2;3)E(da).
The distal half of the salivary gland turns but the proximal half does not in embryos homozygous for Df(3L)BSC13 by stage 14.
The Df(3L)BSC13 chromosome acts as a dominant weak suppressor of telomeric silencing (assayed using the effect of the chromosome on the eye colour phenotype of flies carrying "P{wvar}KR3-2", a stable "brown-red" variant of the P{3'WP-2,wvar}2Lt insertion).
Heterozygosity for Df(3L)BSC13 results in 50.8% X chromosome nondisjunction and 11.9% fourth chromosome nondisjunction in In(1)FM7/X ; svspa-pol females.
The miniwhite markers from both P{lacW}Nmtj1C7 and P{EP}ergic53EP3212 were deleted or disrupted.